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Infantile-onset pompe disease: a tale of two cases

Part of: Metabolic

Published online by Cambridge University Press:  27 January 2020

Drishti Tolani
Affiliation:
Division of Pediatric Cardiology, The Children’s Hospital of Michigan, Detroit, MI, USA
Neha Bansal*
Affiliation:
Division of Pediatric Cardiology, Children’s Hospital at Montefiore, Bronx, NY, USA
Swati Sehgal
Affiliation:
Division of Pediatric Cardiology, The Children’s Hospital of Michigan, Detroit, MI, USA
*
Author for correspondence: Neha Bansal, Division of Pediatric Cardiology, Children’s Hospital at Montefiore, 3415 Bainbridge Ave- R1, Bronx, NY10467, USA. Tel: 718-741-2313; Fax: (718) 920-4351; E-mail: nbansal@montefiore.org
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Abstract

Pompe disease is a type-II glycogen storage disease, and clinical manifestations include hypertrophic cardiomyopathy and generalised muscular hypotonia. Enzyme replacement therapy has proven to be effective in reversing the ventricular hypertrophy. The outcomes are variable depending on time to diagnosis and severity of the cardiac disease. We describe two contrasting cases of patients with infantile-onset Pompe disease. The first child was diagnosed late and had severe cardiac hypertrophy with respiratory decompensation and ventilator dependence and eventual death. The second case was diagnosed at birth with early initiation of therapy resulting in a good outcome. Our cases highlight the importance of early initiation of enzyme replacement therapy to improve clinical outcomes.

Information

Type
Brief Report
Copyright
© Cambridge University Press 2020
Figure 0

Figure 1. Electrocardiogram demonstrating severe biventricular hypertrophy with normal sinus rhythm, depolarisation abnormalities consistent with high voltages, prominent R wave in precordial leads, as well as ST abnormality and T wave inversion in inferolateral leads.

Figure 1

Figure 2. 2D-echocardiogram images ((a) apical view and (b) parasternal view) demonstrating moderate biventricular hypertrophy. LVIDd 2 cm (z score 1.2), LVPWd 0.48 cm (z score 2.9), IVSd 0.56 cm (z score 2.1), ejection fraction 49%. Follow-up images ((c) apical view and (d) parasternal view) at 17 months of age with biweekly enzyme replacement therapy demonstrating resolution of biventricular hypertrophy. LVIDd 3.1 cm (z score 1.7), LVPWd 0.39 cm (z score 0.35), IVSd 0.43 cm (z score 0.00), ejection fraction 64%. LVIDd = left ventricular internal dimension at end-diastole; LVPWd = left ventricular posterior wall thickness at end diastole; IVSd = interventricular septum thickness at end diastole; EF = ejection fraction. The above images were captured during diastole.