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A Novel Approach for Pathway Analysis of GWAS Data Highlights Role of BMP Signaling and Muscle Cell Differentiation in Colorectal Cancer Susceptibility – Erratum

Published online by Cambridge University Press:  23 February 2017

Aniket Mishra  and
Stuart MacGregor
Aniket Mishra
Affiliation:
Genetics and Epidemiology of Colorectal Cancer Consortium (GECCO) and the Colorectal Cancer Family Registry (CCFR), and Statistical Genetics, QIMR Berghofer Medical Research Institute, Brisbane, Queensland, Australia
Stuart MacGregor
Affiliation:
Genetics and Epidemiology of Colorectal Cancer Consortium (GECCO) and the Colorectal Cancer Family Registry (CCFR), and Statistical Genetics, QIMR Berghofer Medical Research Institute, Brisbane, Queensland, Australia

Extract

The publishers regret to announce that the affiliation for the above paper was incorrectly inserted. The correct affiliation is below:

Aniket Mishra1, Genetics and Epidemiology of Colorectal Cancer Consortium (GECCO) and the Colorectal Cancer Family Registry (CCFR), and Stuart MacGregor1

1 Statistical Genetics, QIMR Berghofer Medical Research Institute, Brisbane, Queensland, Australia

Information

Type
Erratum
Information
Twin Research and Human Genetics , Volume 20 , Issue 2 , April 2017 , pp. 186
Copyright
Copyright © The Author(s) 2017 

The publishers regret to announce that the affiliation for the above paper was incorrectly inserted. The correct affiliation is below:

Aniket Mishra1, Genetics and Epidemiology of Colorectal Cancer Consortium (GECCO) and the Colorectal Cancer Family Registry (CCFR), and Stuart MacGregor1

1 Statistical Genetics, QIMR Berghofer Medical Research Institute, Brisbane, Queensland, Australia

References

Mishra, A., Genetics, & Epidemiology of Colorectal Cancer Consortium (GECCO), the Colorectal Cancer Family Registry (CCFR), & MacGregor, S. (2017) A novel approach for pathway analysis of GWAS data highlights role of BMP signaling and muscle cell differentiation in colorectal cancer susceptibility. Journal of Twin Research and Human Genetics, 20, 1–9.Google Scholar