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Depression and bipolar disorder are the most common mood disorders. Recognizing how the mood system works more broadly is foundational for understanding these disorders. While emotions tend to be short-lived, somewhat automatic reactions to specific external stimuli, moods last longer, are caused by several factors, and tend to have more all-encompassing effects on motivation and behavior. An evolutionary behavioral perspective on the mood system views mood as a dynamic psychobehavioral regulatory mechanism, calibrated according to the inputs it receives from the environment in order to produce behavioral outputs that more adaptively fit the perceived balance between rewards and punishments in one’s environment. In depression, mood becomes unnaturally low, leading to a distorted perception of fewer rewards and more frequent punishments in the environment. The psychophysiological stress system, including the hypothalamic–pituitary–adrenal (HPA) axis, is one proximate mechanism that mediates the relationship between environmental inputs (e.g., those that cause stress) and changes in mood. This system might be overactive in people living in contemporary environments that have many evolutionarily novel stressors. This creates the physiological basis for the way in which major individual stressors as well as chronic stress are implicated in the pathophysiology of depression in contemporary Homo sapiens.
As an integral ingredient of human sociality, prosocial behavior requires learning what acts can benefit or harm others. However, it remains unknown how individuals adjust prosocial learning to avoid punishment or to pursue reward. Given that arginine vasopressin (AVP) is a neuropeptide that has been involved in modulating various social behaviors in mammals, it could be a crucial neurochemical facilitator that supports prosocial learning.
Methods
In 50 placebo controls and 54 participants with AVP administration, we examined the modulation of AVP on the prosocial learning characterized by reward and punishment framework, as well as its underlying neurocomputational mechanisms combining computational modeling, event-related potentials and oscillations.
Results
We found a self-bias that individuals learn to avoid punishment asymmetrically more severely than reward-seeking. Importantly, AVP increased behavioral performances and learning rates when making decisions to avoid losses for others and to obtain gains for self. These behavioral effects were underpinned by larger responses of stimulus-preceding negativity (SPN) to anticipation, as well as higher punishment-related feedback-related negativity (FRN) for prosocial learning and reward-related P300 for proself benefits, while FRN and P300 neural processes were integrated into theta (4–7 Hz) oscillation at the outcome evaluation stage.
Conclusions
These results suggest that AVP context-dependently up-regulates altruism for concerning others' losses and reward-seeking for self-oriented benefits. Our findings provide insight into the selectively modulatory roles of AVP in prosocial behaviors depending on learning contexts between proself reward-seeking and prosocial punishment-avoidance.
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